Genes & Development -- Table of Contents (July 15 2002, 16 [14])

QUICK SEARCH:

[advanced]

	Author:		Keyword(s):
Year:		Vol:		Page:

Receive this page by email each issue: [Sign up for eTOCs]

Volume 16, Issue 14: July 15, 2002 [Index by Author] [Cover Caption]

Other Issues:

Perspectives

Research Communications

Research Papers

Find articles in this issue containing these words:
[Search ALL Issues]

To see an article, click its [Full Text] link. To review many abstracts, check the boxes to the left of the titles you want, and click the 'Get All Checked Abstract(s)' button. To see one abstract at a time, click its [Abstract] link.

Perspectives:

Mary Grace Goll and Timothy H. Bestor: Histone modification and replacement in chromatin activation
Genes Dev. 2002 16: 1739-1742. [Full Text] [PDF]

Lynne E. Maquat: NASty effects on fibrillin pre-mRNA splicing: another case of ESE does it, but proposals for translation-dependent splice site choice live on
Genes Dev. 2002 16: 1743-1753. [Full Text] [PDF]

Research Communications:

Massimo Caputi, Raymond J. Kendzior, Jr., and Karen L. Beemon: A nonsense mutation in the fibrillin-1 gene of a Marfan syndrome patient induces NMD and disrupts an exonic splicing enhancer
Genes Dev. 2002 16: 1754-1759. [Abstract] [Full Text] [PDF]

Yoshinobu Sugitani, Shigeyasu Nakai, Osamu Minowa, Miyuki Nishi, Kou-ichi Jishage, Hitoshi Kawano, Kensaku Mori, Masaharu Ogawa, and Tetsuo Noda: Brn-1 and Brn-2 share crucial roles in the production and positioning of mouse neocortical neurons
Genes Dev. 2002 16: 1760-1765. [Abstract] [Full Text] [PDF] Supplemental Research Data

Research Papers:

Hiromi Nakagawa, Joon-Kyu Lee, Jerard Hurwitz, Robin C. Allshire, Jun-ichi Nakayama, Shiv I.S. Grewal, Katsunori Tanaka, and Yota Murakami: Fission yeast CENP-B homologs nucleate centromeric heterochromatin by promoting heterochromatin-specific histone tail modifications
Genes Dev. 2002 16: 1766-1778. [Abstract] [Full Text] [PDF]

Makoto Tachibana, Kenji Sugimoto, Masami Nozaki, Jun Ueda, Tsutomu Ohta, Misao Ohki, Mikiko Fukuda, Naoki Takeda, Hiroyuki Niida, Hiroyuki Kato, and Yoichi Shinkai: G9a histone methyltransferase plays a dominant role in euchromatic histone H3 lysine 9 methylation and is essential for early embryogenesis
Genes Dev. 2002 16: 1779-1791. [Abstract] [Full Text] [PDF]

Jean-Bernard Lazaro, Peter J. Bailey, and Andrew B. Lassar: Cyclin D-cdk4 activity modulates the subnuclear localization and interaction of MEF2 with SRC-family coactivators during skeletal muscle differentiation
Genes Dev. 2002 16: 1792-1805. [Abstract] [Full Text] [PDF]

Stephan P. Persengiev, Laxminarayana R. Devireddy, and Michael R. Green: Inhibition of apoptosis by ATFx: a novel role for a member of the ATF/CREB family of mammalian bZIP transcription factors
Genes Dev. 2002 16: 1806-1814. [Abstract] [Full Text] [PDF]

Robyn M. Howard and Meera V. Sundaram: C. elegans EOR-1/PLZF and EOR-2 positively regulate Ras and Wnt signaling and function redundantly with LIN-25 and the SUR-2 Mediator component
Genes Dev. 2002 16: 1815-1827. [Abstract] [Full Text] [PDF]

Elisabeth Saller, Ann Kelley, and Mariann Bienz: The transcriptional repressor Brinker antagonizes Wingless signaling
Genes Dev. 2002 16: 1828-1838. [Abstract] [Full Text] [PDF]

Dagmar Wilhelm and Christoph Englert: The Wilms tumor suppressor WT1 regulates early gonad development by activation of Sf1
Genes Dev. 2002 16: 1839-1851. [Abstract] [Full Text] [PDF]

Kristina M. Johnson, Jin Wang, Andrea Smallwood, Charina Arayata, and Michael Carey: TFIID and human mediator coactivator complexes assemble cooperatively on promoter DNA
Genes Dev. 2002 16: 1852-1863. [Abstract] [Full Text] [PDF]

Cover Caption:

Cover

The mammalian euchromatic histone H3 Lys 9 methyltransferase, G9a, is essential for normal embryonic development. Shown hereare DAPI-stained transverse sections from an E8.5 wild-type (background)and an E9.5 G9a^/ (inset) mouse embryo. By E9.5, G9a^/ mouse embryos display severe developmental retardation, includingincomplete anterior neural groove fusion (shown here), and earlylethality. Mitotic cells are present in similar ratios betweenE9.5 G9a^/ and wild-type mouse embryos. However, a drastic accumulationof apoptotic cells is commonly observed in G9a^/ embryos, suggesting that the G9a^/ embryonic growth defect is, at least in part, due to apoptoticcell death. (For details, see Tachibana et al., p. 1779.)