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Volume 17, Issue 12:  June 15, 2003  [Index by Author]  [Cover Caption] 

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  Research Communications
  Research Papers
  Errata
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Reviews:

Amparo Palmer and Rüdiger Klein
Multiple roles of ephrins in morphogenesis, neuronal networking, and brain function
Genes Dev. 2003 17: 1429-1450. [Full Text] [PDF]  

Research Communications:

Yasumasa Bessho, Hiromi Hirata, Yoshito Masamizu, and Ryoichiro Kageyama
Periodic repression by the bHLH factor Hes7 is an essential mechanism for the somite segmentation clock
Genes Dev. 2003 17: 1451-1456. Published in Advance June 3, 2003, 10.1101/gad.1092303 [Abstract] [Full Text] [PDF]  

Joyce Tay, Rebecca Hodgman, Madathia Sarkissian, and Joel D. Richter
Regulated CPEB phosphorylation during meiotic progression suggests a mechanism for temporal control of maternal mRNA translation
Genes Dev. 2003 17: 1457-1462. [Abstract] [Full Text] [PDF]  

J. Paul Taylor, Addis A. Taye, Catherine Campbell, Parsa Kazemi-Esfarjani, Kenneth H. Fischbeck, and Kyung-Tai Min
Aberrant histone acetylation, altered transcription, and retinal degeneration in a Drosophila model of polyglutamine disease are rescued by CREB-binding protein
Genes Dev. 2003 17: 1463-1468. [Abstract] [Full Text] [PDF] Supplemental Research Data  

Karim Sorefan, Jon Booker, Karine Haurogné, Magali Goussot, Katherine Bainbridge, Eloise Foo, Steven Chatfield, Sally Ward, Christine Beveridge, Catherine Rameau, and Ottoline Leyser
MAX4 and RMS1 are orthologous dioxygenase-like genes that regulate shoot branching in Arabidopsis and pea
Genes Dev. 2003 17: 1469-1474. [Abstract] [Full Text] [PDF] Supplemental Research Data  

Research Papers:

Deepak Nijhawan, Min Fang, Elie Traer, Qing Zhong, Wenhua Gao, Fenghe Du, and Xiaodong Wang
Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation
Genes Dev. 2003 17: 1475-1486. Published in Advance June 3, 2003, 10.1101/gad.1093903 [Abstract] [Full Text] [PDF]  

Qi-Heng Yang, Robin Church-Hajduk, Jinyu Ren, Michelle L. Newton, and Chunying Du
Omi/HtrA2 catalytic cleavage of inhibitor of apoptosis (IAP) irreversibly inactivates IAPs and facilitates caspase activity in apoptosis
Genes Dev. 2003 17: 1487-1496. [Abstract] [Full Text] [PDF]  

Yasushi Takeuchi, Takashi Horiuchi, and Takehiko Kobayashi
Transcription-dependent recombination and the role of fork collision in yeast rDNA
Genes Dev. 2003 17: 1497-1506. Published in Advance June 3, 2003, 10.1101/gad.1085403 [Abstract] [Full Text] [PDF]  

Fiona H. MacIver, Kayoko Tanaka, Alasdair M. Robertson, and Iain M. Hagan
Physical and functional interactions between polo kinase and the spindle pole component Cut12 regulate mitotic commitment in S. pombe
Genes Dev. 2003 17: 1507-1523. [Abstract] [Full Text] [PDF]  

Kirsten R. Benjamin, Chao Zhang, Kevan M. Shokat, and Ira Herskowitz
Control of landmark events in meiosis by the CDK Cdc28 and the meiosis-specific kinase Ime2
Genes Dev. 2003 17: 1524-1539. Published in Advance June 3, 2003, 10.1101/gad.1101503 [Abstract] [Full Text] [PDF]  

Claudia Köhler, Lars Hennig, Charles Spillane, Stephane Pien, Wilhelm Gruissem, and Ueli Grossniklaus
The Polycomb-group protein MEDEA regulates seed development by controlling expression of the MADS-box gene PHERES1
Genes Dev. 2003 17: 1540-1553. [Abstract] [Full Text] [PDF] Supplemental Research Data  

Errata:

Errata for vol. 17, p. 959
Genes Dev. 2003 17: 1554. [Full Text] [PDF]  

To see an article, click its [Full Text] link. To review many abstracts, check the boxes to the left of the titles you want, and click the 'Get All Checked Abstract(s)' button. To see one abstract at a time, click its [Abstract] link.


Cover Caption:

Cover Cover The type I MADS-box gene PHERES1 is a primary target of the Polycomb group protein MEDEA in Arabidopsis. Shown here is a wild-type, heart-stage Arabidopsis seed expressing a PHERES1::-GLUCURONIDASE transgene. After fertilization, PHERES1 is transiently expressed in the embryo and the endosperm. When seeds reach the heart stage, PHERES1 expression becomes restricted to the chalazal region of the endosperm (shown here). In medea mutant seeds, PHERES1 is highly expressed until the seeds abort. (For details, see Kohler et al., p. 1540.)


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