Genes & Development -- Table of Contents (April 15 2003, 17 [8])

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Volume 17, Issue 8: April 15, 2003 [Index by Author] [Cover Caption]

Other Issues:

Reviews

Research Communications

Research Papers

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Reviews:

Jeffrey L. Goldberg: How does an axon grow?
Genes Dev. 2003 17: 941-958. [Full Text] [PDF]

Research Communications:

Cornelis F. Calkhoven, Christine Müller, Richard Martin, Goradz Krosl, Trang Hoang, and Achim Leutz: Translational control of SCL-isoform expression in hematopoietic lineage choice
Genes Dev. 2003 17: 959-964. [Abstract] [Full Text] [PDF]

Xin Wang, Lee Zou, Huyong Zheng, Qingyi Wei, Stephen J. Elledge, and Lei Li: Genomic instability and endoreduplication triggered by RAD17 deletion
Genes Dev. 2003 17: 965-970. Published in Advance April 2, 2003, 10.1101/gad.1065103 [Abstract] [Full Text] [PDF] Supplemental Research Data

Hang Shi and Rui-Ming Xu: Crystal structure of the Drosophila Mago nashi-Y14 complex
Genes Dev. 2003 17: 971-976. [Abstract] [Full Text] [PDF]

Research Papers:

John Gately Luz, Christian A. Hassig, Catherine Pickle, Adam Godzik, Barbara J. Meyer, and Ian A. Wilson: XOL-1, primary determinant of sexual fate in C. elegans, is a GHMP kinase family member and a structural prototype for a class of developmental regulators
Genes Dev. 2003 17: 977-990. Published in Advance April 2, 2003, 10.1101/gad.1082303 [Abstract] [Full Text] [PDF]

Lee P. Lim, Nelson C. Lau, Earl G. Weinstein, Aliaa Abdelhakim, Soraya Yekta, Matthew W. Rhoades, Christopher B. Burge, and David P. Bartel: The microRNAs of Caenorhabditis elegans
Genes Dev. 2003 17: 991-1008. Published in Advance April 2, 2003, 10.1101/gad.1074403 [Abstract] [Full Text] [PDF] Supplemental Research Data

Tomoyuki Sawado, Jessica Halow, M.A. Bender, and Mark Groudine: The $beta$ -globin locus control region (LCR) functions primarily by enhancing the transition from transcription initiation to elongation
Genes Dev. 2003 17: 1009-1018. Published in Advance April 2, 2003, 10.1101/gad.1072303 [Abstract] [Full Text] [PDF] Supplemental Research Data

Natalie Rezai-Zadeh, Xiaohong Zhang, Fares Namour, Gyorgy Fejer, Yu-Der Wen, Ya-Li Yao, Ildiko Gyory, Kenneth Wright, and Edward Seto: Targeted recruitment of a histone H4-specific methyltransferase by the transcription factor YY1
Genes Dev. 2003 17: 1019-1029. [Abstract] [Full Text] [PDF]

Xiaoyuan He, Asad U. Khan, Hailing Cheng, Donald L. Pappas, Jr., Michael Hampsey, and Claire L. Moore: Functional interactions between the transcription and mRNA 3' end processing machineries mediated by Ssu72 and Sub1
Genes Dev. 2003 17: 1030-1042. [Abstract] [Full Text] [PDF]

Viswanathan Chinnusamy, Masaru Ohta, Siddhartha Kanrar, Byeong-ha Lee, Xuhui Hong, Manu Agarwal, and Jian-Kang Zhu: ICE1: a regulator of cold-induced transcriptome and freezing tolerance in Arabidopsis
Genes Dev. 2003 17: 1043-1054. Published in Advance April 2, 2003, 10.1101/gad.1077503 [Abstract] [Full Text] [PDF] Supplemental Research Data

Takashi Soyano, Ryuichi Nishihama, Kayoko Morikiyo, Masaki Ishikawa, and Yasunori Machida: NQK1/NtMEK1 is a MAPKK that acts in the NPK1 MAPKKK-mediated MAPK cascade and is required for plant cytokinesis
Genes Dev. 2003 17: 1055-1067. [Abstract] [Full Text] [PDF]

Cover Caption:

Cover

The structure of XOL-1, the primary determinant of sexual fate in C. elegans. Shown here is a ribbon diagram of the XOL-1crystal structure (red). XOL-1 is merged with a Nomarski photomicrographof a C. elegans male and hermaphrodite mating pair (cyan falsecolor). These worms have a defective dpy-28 dosage compensationgene that prevents hermaphrodites from down-regulating their X-linkedtranscript levels and causes death or a Dumpy phenotype (shownhere). The background of the image is the diffraction patterngenerated from crystals of the XOL-1 protein. When active, XOL-1promotes male development by ensuring that dpy-28 and other hermaphrodite-specificdosage compensation and/or sex determination genes are functionallyinactive; when inactive, XOL-1 promotes the hermaphrodite fate,including the activation of X-chromosome dosage compensation.The crystal structure unexpectedly showed XOL-1 to be a memberof the GHMP kinase family, previously characterized as comprisingonly small molecule kinases involved in metabolic pathways. (Fordetails, see Luz et al., p. 977.)